mouse anti alpha tubulin monoclonal antibody Search Results


96
LI-COR mouse α actin
Mouse α Actin, supplied by LI-COR, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Valiant Co Ltd actin mp biomedicals 08637931
Actin Mp Biomedicals 08637931, supplied by Valiant Co Ltd, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Boster Bio monoclonal mouse anti glial fibrillary acidic protein antibody
Fig. 2. (A–C) Photomicrographs of rats' cerebellar cortex stained with <t>anti-GFAP</t> antibodies (X 1000). (A) Represents 12M-group, with mild GFAP immunoreaction while (B) and (C) represents 24M (moderate reaction) and 32M (sever reaction) groups respectively. Note: GFAP positive cells (arrow) appeared with dark brown discoloration. (D) Represents GFAP immunoreaction changes in different age groups. * statistically significant (p < 0.05) difference in comparison to 12M-group. ** statistically significant (p < 0.05) difference in comparison to 24M-group. Data are presented as mean ± standard deviation, (n = 30). (For interpretation of the references to color in this figure legend, the reader is referred to the Web version of this article.)
Monoclonal Mouse Anti Glial Fibrillary Acidic Protein Antibody, supplied by Boster Bio, used in various techniques. Bioz Stars score: 88/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Boster Bio primary antibody mouse monoclonal anti mouse α actinin
Fig. 2. (A–C) Photomicrographs of rats' cerebellar cortex stained with <t>anti-GFAP</t> antibodies (X 1000). (A) Represents 12M-group, with mild GFAP immunoreaction while (B) and (C) represents 24M (moderate reaction) and 32M (sever reaction) groups respectively. Note: GFAP positive cells (arrow) appeared with dark brown discoloration. (D) Represents GFAP immunoreaction changes in different age groups. * statistically significant (p < 0.05) difference in comparison to 12M-group. ** statistically significant (p < 0.05) difference in comparison to 24M-group. Data are presented as mean ± standard deviation, (n = 30). (For interpretation of the references to color in this figure legend, the reader is referred to the Web version of this article.)
Primary Antibody Mouse Monoclonal Anti Mouse α Actinin, supplied by Boster Bio, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Boster Bio pik3ca
Figure 3. (A) <t>PIK3CA</t> immunoreactivity in angiosarcoma of the scalp and face and hemangioma. Angiosarcoma lesions exhibited significantly higher PIK3CA immunoreactivity than hemangioma (x2=20.97, P=.001, P<.01). (B) Representative images of immunohistochemical staining of PIK3CA in (a) angiosarcoma and (b) hemangioma tissues (SP, 100). PIK3CA=phospha- tidylinositol-4,5-bisphosphate 3-kinase catalytic subunit alpha.
Pik3ca, supplied by Boster Bio, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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OriGene cryaa origene cf505577
Figure 3. (A) <t>PIK3CA</t> immunoreactivity in angiosarcoma of the scalp and face and hemangioma. Angiosarcoma lesions exhibited significantly higher PIK3CA immunoreactivity than hemangioma (x2=20.97, P=.001, P<.01). (B) Representative images of immunohistochemical staining of PIK3CA in (a) angiosarcoma and (b) hemangioma tissues (SP, 100). PIK3CA=phospha- tidylinositol-4,5-bisphosphate 3-kinase catalytic subunit alpha.
Cryaa Origene Cf505577, supplied by OriGene, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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OriGene cryab origene cf500680
Figure 3. (A) <t>PIK3CA</t> immunoreactivity in angiosarcoma of the scalp and face and hemangioma. Angiosarcoma lesions exhibited significantly higher PIK3CA immunoreactivity than hemangioma (x2=20.97, P=.001, P<.01). (B) Representative images of immunohistochemical staining of PIK3CA in (a) angiosarcoma and (b) hemangioma tissues (SP, 100). PIK3CA=phospha- tidylinositol-4,5-bisphosphate 3-kinase catalytic subunit alpha.
Cryab Origene Cf500680, supplied by OriGene, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Boster Bio mouse monoclonal anti s100β antibody
Spinal cord astrocyte identification and high mobility group box-1 (HMGB1) knockdown. a Spinal cord astrocytes were identified using immunofluorescence. The percentage of cells stained with the astrocytic marker <t>S100β,</t> which were identified as astrocytes, was more than 95% of the total cells (three replicates). b HMGB1 knockdown efficiency in the plasma membrane and cytoplasm of spinal cord astrocytes was evaluated using Western blot for HMGB1 protein levels. Results were obtained after 72 h of specific HMGB1 shRNA treatment. HMGB1 protein levels were decreased to approximately 30% of normal levels with shRNA multiplicity of infection 60 as compared to normal astrocytes. * P < 0.05 vs. normal group (three replicates)
Mouse Monoclonal Anti S100β Antibody, supplied by Boster Bio, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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86
Aviva Systems mouse monoclonal antibody against α tubulin
Spinal cord astrocyte identification and high mobility group box-1 (HMGB1) knockdown. a Spinal cord astrocytes were identified using immunofluorescence. The percentage of cells stained with the astrocytic marker <t>S100β,</t> which were identified as astrocytes, was more than 95% of the total cells (three replicates). b HMGB1 knockdown efficiency in the plasma membrane and cytoplasm of spinal cord astrocytes was evaluated using Western blot for HMGB1 protein levels. Results were obtained after 72 h of specific HMGB1 shRNA treatment. HMGB1 protein levels were decreased to approximately 30% of normal levels with shRNA multiplicity of infection 60 as compared to normal astrocytes. * P < 0.05 vs. normal group (three replicates)
Mouse Monoclonal Antibody Against α Tubulin, supplied by Aviva Systems, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Boster Bio mouse anti ibv n protein monoclonal antibody
Spinal cord astrocyte identification and high mobility group box-1 (HMGB1) knockdown. a Spinal cord astrocytes were identified using immunofluorescence. The percentage of cells stained with the astrocytic marker <t>S100β,</t> which were identified as astrocytes, was more than 95% of the total cells (three replicates). b HMGB1 knockdown efficiency in the plasma membrane and cytoplasm of spinal cord astrocytes was evaluated using Western blot for HMGB1 protein levels. Results were obtained after 72 h of specific HMGB1 shRNA treatment. HMGB1 protein levels were decreased to approximately 30% of normal levels with shRNA multiplicity of infection 60 as compared to normal astrocytes. * P < 0.05 vs. normal group (three replicates)
Mouse Anti Ibv N Protein Monoclonal Antibody, supplied by Boster Bio, used in various techniques. Bioz Stars score: 91/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Valiant Co Ltd mouse antibody
Spinal cord astrocyte identification and high mobility group box-1 (HMGB1) knockdown. a Spinal cord astrocytes were identified using immunofluorescence. The percentage of cells stained with the astrocytic marker <t>S100β,</t> which were identified as astrocytes, was more than 95% of the total cells (three replicates). b HMGB1 knockdown efficiency in the plasma membrane and cytoplasm of spinal cord astrocytes was evaluated using Western blot for HMGB1 protein levels. Results were obtained after 72 h of specific HMGB1 shRNA treatment. HMGB1 protein levels were decreased to approximately 30% of normal levels with shRNA multiplicity of infection 60 as compared to normal astrocytes. * P < 0.05 vs. normal group (three replicates)
Mouse Antibody, supplied by Valiant Co Ltd, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Boster Bio mouse monoclonal anti α sma
Spinal cord astrocyte identification and high mobility group box-1 (HMGB1) knockdown. a Spinal cord astrocytes were identified using immunofluorescence. The percentage of cells stained with the astrocytic marker <t>S100β,</t> which were identified as astrocytes, was more than 95% of the total cells (three replicates). b HMGB1 knockdown efficiency in the plasma membrane and cytoplasm of spinal cord astrocytes was evaluated using Western blot for HMGB1 protein levels. Results were obtained after 72 h of specific HMGB1 shRNA treatment. HMGB1 protein levels were decreased to approximately 30% of normal levels with shRNA multiplicity of infection 60 as compared to normal astrocytes. * P < 0.05 vs. normal group (three replicates)
Mouse Monoclonal Anti α Sma, supplied by Boster Bio, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Fig. 2. (A–C) Photomicrographs of rats' cerebellar cortex stained with anti-GFAP antibodies (X 1000). (A) Represents 12M-group, with mild GFAP immunoreaction while (B) and (C) represents 24M (moderate reaction) and 32M (sever reaction) groups respectively. Note: GFAP positive cells (arrow) appeared with dark brown discoloration. (D) Represents GFAP immunoreaction changes in different age groups. * statistically significant (p < 0.05) difference in comparison to 12M-group. ** statistically significant (p < 0.05) difference in comparison to 24M-group. Data are presented as mean ± standard deviation, (n = 30). (For interpretation of the references to color in this figure legend, the reader is referred to the Web version of this article.)

Journal: Translational Research in Anatomy

Article Title: JAK-1/STAT-3 pathway mediated role in aging cerebellar cortex degenerative changes of albino wistar rats

doi: 10.1016/j.tria.2020.100089

Figure Lengend Snippet: Fig. 2. (A–C) Photomicrographs of rats' cerebellar cortex stained with anti-GFAP antibodies (X 1000). (A) Represents 12M-group, with mild GFAP immunoreaction while (B) and (C) represents 24M (moderate reaction) and 32M (sever reaction) groups respectively. Note: GFAP positive cells (arrow) appeared with dark brown discoloration. (D) Represents GFAP immunoreaction changes in different age groups. * statistically significant (p < 0.05) difference in comparison to 12M-group. ** statistically significant (p < 0.05) difference in comparison to 24M-group. Data are presented as mean ± standard deviation, (n = 30). (For interpretation of the references to color in this figure legend, the reader is referred to the Web version of this article.)

Article Snippet: Monoclonal mouse anti-glial fibrillary acidic protein antibody (1:20 dilution) and Anti-active caspase-3 rabbit monoclonal antibody (1:200 dilution) (Boster Biological Technology inc. ltd., USA) were mounted to the sections and incubated overnight at 4° c in a humidified chamber.

Techniques: Staining, Comparison, Standard Deviation

Figure 3. (A) PIK3CA immunoreactivity in angiosarcoma of the scalp and face and hemangioma. Angiosarcoma lesions exhibited significantly higher PIK3CA immunoreactivity than hemangioma (x2=20.97, P=.001, P<.01). (B) Representative images of immunohistochemical staining of PIK3CA in (a) angiosarcoma and (b) hemangioma tissues (SP, 100). PIK3CA=phospha- tidylinositol-4,5-bisphosphate 3-kinase catalytic subunit alpha.

Journal: Medicine

Article Title: Aberrant PTEN, PIK3CA, pMAPK, and TP53 expression in human scalp and face angiosarcoma

doi: 10.1097/md.0000000000026779

Figure Lengend Snippet: Figure 3. (A) PIK3CA immunoreactivity in angiosarcoma of the scalp and face and hemangioma. Angiosarcoma lesions exhibited significantly higher PIK3CA immunoreactivity than hemangioma (x2=20.97, P=.001, P<.01). (B) Representative images of immunohistochemical staining of PIK3CA in (a) angiosarcoma and (b) hemangioma tissues (SP, 100). PIK3CA=phospha- tidylinositol-4,5-bisphosphate 3-kinase catalytic subunit alpha.

Article Snippet: The slides were incubated overnight at 4°C with a rabbit polyclonal antibody against human PTEN, PIK3CA, or pMAPK (1:100; Wuhan Boster Biological Technology, Ltd., Wuhan, China), or a mouse monoclonal antibody against human TP53 (1:100; MXB Biotechnologies, Fuzhou, China).

Techniques: Immunohistochemical staining, Staining

Spinal cord astrocyte identification and high mobility group box-1 (HMGB1) knockdown. a Spinal cord astrocytes were identified using immunofluorescence. The percentage of cells stained with the astrocytic marker S100β, which were identified as astrocytes, was more than 95% of the total cells (three replicates). b HMGB1 knockdown efficiency in the plasma membrane and cytoplasm of spinal cord astrocytes was evaluated using Western blot for HMGB1 protein levels. Results were obtained after 72 h of specific HMGB1 shRNA treatment. HMGB1 protein levels were decreased to approximately 30% of normal levels with shRNA multiplicity of infection 60 as compared to normal astrocytes. * P < 0.05 vs. normal group (three replicates)

Journal: Journal of Neuroinflammation

Article Title: Inhibition of HMGB1 reduces rat spinal cord astrocytic swelling and AQP4 expression after oxygen-glucose deprivation and reoxygenation via TLR4 and NF-κB signaling in an IL-6-dependent manner

doi: 10.1186/s12974-017-1008-1

Figure Lengend Snippet: Spinal cord astrocyte identification and high mobility group box-1 (HMGB1) knockdown. a Spinal cord astrocytes were identified using immunofluorescence. The percentage of cells stained with the astrocytic marker S100β, which were identified as astrocytes, was more than 95% of the total cells (three replicates). b HMGB1 knockdown efficiency in the plasma membrane and cytoplasm of spinal cord astrocytes was evaluated using Western blot for HMGB1 protein levels. Results were obtained after 72 h of specific HMGB1 shRNA treatment. HMGB1 protein levels were decreased to approximately 30% of normal levels with shRNA multiplicity of infection 60 as compared to normal astrocytes. * P < 0.05 vs. normal group (three replicates)

Article Snippet: Primary antibodies include rabbit polyclonal anti-HMGB1 antibody for rat, mouse, and human (Abcam, Cat# ab18256, RRID:AB_444360, Cambridge, UK); rabbit polyclonal anti-AQP4 antibody for rat, mouse, human, and pig (Abcam, Cat# ab46182, RRID: AB_955676); mouse monoclonal anti-TLR4 antibody for rat, mouse, human, pig, baboon, bovine, and Chinese hamster (Novus, Cat# 76B357.1, RRID: AB_839000, Littleton, CO, USA); rabbit polyclonal anti-TLR4 antibody for rat, mouse, human, and rabbit (Boster, Cat# BA1717, RRID:AB_2716293); rabbit polyclonal anti-myeloid differentiation primary response gene 88 (MyD88) antibody for rat and human (Abcam, Cat# ab131071, RRID: AB_11156885); mouse monoclonal anti-IκBα antibody for rat, mouse, human, monkey, bovine, pig, and guinea pig (Cell Signaling Technology, Cat# 4814, RRID: AB_390781, Boston, MA, USA); mouse monoclonal anti-p-IκBα antibody for rat, mouse, human, and monkey (Cell Signaling Technology, Cat# 9246, RRID:AB_2267145); rabbit monoclonal anti-NF-κB antibody for rat, mouse, human, monkey, and bovine (Cell Signaling Technology, Cat# 4764, RRID:AB_823578); mouse monoclonal anti-GAPDH antibody for rat, mouse, and human (Beyotime, Cat# AF0006, RRID: AB_2715590, Shanghai, China); mouse monoclonal anti-Histone H3 antibody for rat, mouse, and human (Beyotime, Cat# AF0009, RRID: AB_2715593); and mouse monoclonal anti-S100β antibody for rat, mouse, human, rabbit, and pig (Boster, Cat# BM0120, RRID:AB_2716291).

Techniques: Knockdown, Immunofluorescence, Staining, Marker, Clinical Proteomics, Membrane, Western Blot, shRNA, Infection